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I am also attached to the programme because of the wonderful people that comprise the SCID Panel of Experts, a large group of leading international experts in conflict resolution and peacebuilding. I am very attached to it for this reason and also because of the inspirational students I had the pleasure of working with – immensely hard working (mostly working in difficult jobs in conflict-affected environments and still finding time to complete a Master’s degree) dedicated to giving their all to addressing the challenges of conflict and to continue learning and progressing uncomplaining (even when the harsh realities of working in conflict zones hit home) and brilliant in their insights, compassion and commitment. I was solely responsible for developing the SCID programme from scratch over a 2-year period and delivering it since its inception in 2012. Nonetheless, it has been difficult to formally leave SCID. I hope, therefore, that my departure to Monash University will broaden the networks, discussion and action on issues related to security, conflict and international development. However, I intend to maintain this Blog for everyone associated with SCID and for anyone with an interest in conflict resolution, peacebuilding and development. It is difficult to be reminded of the wonderful SCID community we built together, now that I am working on a different programme. To learn more or make an appointment, please call (650) 502-7179.It is with mixed feelings that I write this post to announce changes in this Blog. In a clinical trial, patients receive an antibody before their stem cell transplant to make space for the normal stem cells to grow, without the need for chemotherapy. We are also working to make stem cell transplantation safer for patients with SCID by eliminating the need for chemotherapy before transplant. Bertaina has performed more than 400 transplants using this approach. This means that every patient with SCID can now find a suitable stem cell donor. This technique has been shown to significantly reduce the risk of GVHD to the level seen in fully matched donors, while also reducing other posttransplant complications. Stanford’s Alice Bertaina, MD, PhD, is an internationally renowned expert in depleting T cells from a mismatched stem cell graft to reduce GVHD. We are working to make stem cell transplants safer and accessible to every patient with SCID. Why choose Stanford Medicine Children’s Health for SCID treatment? It usually takes four to six weeks after transplantation for your child to develop adequate immune function to protect them from infections so that they can be discharged from the hospital. If no family member is a suitable donor, a matched unrelated donor can be used. Stanford doctors can now deplete T cells that cause graft-versus-host disease from the stem cell graft before transplantation, reducing the risk of GVHD and enabling every patient to find a donor. In the past, using a mismatched donor could increase the chances of developing graft-versus-host disease (GVHD). If a healthy matched sibling donor is not available, then stem cell transplantation from a half-matched, or haploidentical, family donor, usually the mother, is the next-best option. The therapy of choice for most patients with SCID is stem cell transplantation from a healthy matched donor, usually a sibling. The only curative therapy for SCID is stem cell transplantation from a healthy individual or with the patient’s own genetically modified cells, also known as gene therapy. How is SCID treated at Stanford Medicine Children’s Health? Your child’s diagnosis will be confirmed by genetic testing and an assessment of your child’s T and B cell numbers and function. Patients with SCID are categorized by their primary genetic defect, but common to all forms of SCID is an absence of protective T and B cells (immune cells that help attack invaders like bacteria, viruses, and fungi). Now, your child may have been diagnosed based on a newborn screening before he or she developed any infections. In the past, patients were diagnosed based on their clinical presentation or family history. Symptoms include diarrhea, fungal infections, and poor physical growth. What are the symptoms of SCID, and how is it diagnosed?
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A variety of genetic defects can produce SCID. Patients with severe combined immunodeficiency (SCID) are born without a functioning immune system and therefore are at increased risk of infections and death before the age of 2. What is severe combined immunodeficiency (SCID)?